This may make the postsynaptic membrane depolarized ( less negative ).
2.
These neurotransmitters bind to receptors on the postsynaptic membrane and lead to its depolarization.
3.
For example, curare is a poison that stops acetylcholine from depolarizing the postsynaptic membrane, causing paralysis.
4.
These inhibitory influences ( IPSPs ) are mediated by inhibitory neurotransmitter systems that cause postsynaptic membranes to hyperpolarize.
5.
Immediately behind the postsynaptic membrane is an elaborate complex of interlinked proteins called the postsynaptic density ( PSD ).
6.
Glutamate then binds to AMPA receptors contained in the postsynaptic membrane causing the influx of positively charged sodium atoms.
7.
When a glutamatergic synapse releases glutamate it binds to any AMPA and the NMDA receptors present in the postsynaptic membrane.
8.
Once in the synapse, neurotransmitters will rapidly move across the synapse to attach themselves to receptors on the postsynaptic membrane.
9.
When multiple EPSPs occur on a single patch of postsynaptic membrane, their combined effect is the sum of the individual EPSPs.
10.
The motor neuron then innervates the muscle fibers to contraction by causing an action potential on the postsynaptic membrane of the neuromuscular junction.
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